Speaking from the Heart with Stephen Hussey

Well, hello, ladies and gents Robert Sykes Kido Savage.com. And today, I've got special guest, Stephen hercy back on the podcast. This is the fourth time. I've had him on the show, every single time. I talk to him. I'm like, man. This could be like a three-hour episode because he is just incredibly knowledgeable about all things heart, health diet. He's type 1 diabetics. He brings a very unique approach to nutrition. That just adds a whole nother

level of context there. And I just, I really appreciate who he is, what he's doing, what information is putting on the road. He's got a new book, Coming out on April 7th, and I'm going to be 100% flipping of that thing, cover to cover and learn as much as I can. Because his knowledge of, you know, the heart and heart health. Like I said is just unparalleled. So very, very smart guy. I have no doubt. They will take something from this conversation. So that further Ado, sit back,

relax. Enjoy the podcast with Steven hussy. We are live. Steven has a how are you men doing? Well, how are you? I'm good. I'm good. So I was, I was perusing back in our prior podcast episodes and you've been on the show three other times besides this was just kind of crazy. The first one. I think we mostly do Vin to your four pillars of evolution Evolution.

I think we talked quite a bit about that and then the last two we touched on I think fourth phase water and how blood It's to the the body independent of the pump, you know, the heart as you said, it's not really referred to as a pump. We drove into that. And then I think, in the last episode, that was about a month after your heart attacks. We talked about that. That's more or less.

I think where I didn't listen to all them, that's kind of what we, what we covered in those episodes, but you've got a new book coming out, you've done a lot since that last conversation that we had in February of 2021. So just kind of bring us up to speed man, it for you, but it hasn't listened to any of those episodes which I encourage everybody to do. Can you? It's like a quick little intro or bio into you. Yeah, for sure. So, yeah, I'm Steven has see I'm a chiropractor and functional

medicine practitioner. You know, my health Journey was kind of inspired or my work is inspired by my health Journey because at a very young age, I was diagnosed with a lot of different chronic conditions, you know, everything from from chronic hives to allergies to irritable bowel syndrome and eventually ended up with autoimmune type 1, diabetes. Which heavily predisposes me to heart disease and so throughout all of my schooling and training and then in my own independent

research. I've been really interested in heart disease. And so so I kind of dug into that a lot. I've got quite a lot of information and I decided to put it all down in this book and became especially relevant to me when I had a heart attack, a little over a year ago, at the age of 34 and she's really trying to dive in and see why this happened to me and what I found was pretty shocked. Because you know, first I was like, I have this information is like, why couldn't I prevent

this? But then my experience in the hospital and the care that I was given. I mean, it was it was amazing what they're able to do to save my life when that happened. But after the fact based on everything, I knew I realized that their approach to Healing my heart and preventing it from happening again. Was was very pharmaceutical based and when you look at the research, it's not very good based on that.

And so I wrote out that story in this book as Is like this big introduction to why I'm so interested in heart disease and all the kind of fascinating things that I found. I summarized in this book and realized that, you know, based on the care. I received in the hospital, this it's information that I have. You know, first. I was doubting myself, my God, can't, I can't release this book or whatever, but then I realized that it's even more important.

When you release this information because in my opinion, Western medicine is pretty lost. When it comes to Healing the heart and and preventing these types of things. Yeah. I remember when you sent me. I guess it was like the preliminary text to the book and you cause you had written a lot of the book before the heart attack happened and the heart attack happened. And you were kind of going through this period of, you know, should I write this?

Should I put this out there? I mean, I feel kind of hypocritical, etc. Etc. So you wrote this really eloquent, you know, Memoir kind of showcasing and illustrating all that and I feel like, you know, going in hindsight now, the fact that you've had that heart attack, it just puts your perspective on such another tear that makes everything. You're saying that much more relevant. Yeah, definitely.

And and you know, like I was faced with this decision, like do I, you know, I just had a heart attack, which was, you know, a big deal. Like it was a major heart attack to and like, only twelve percent of people who have that type of heart attack outside of the hospital survive. And so, so I was, you know, very lucky to for those for the people that did save my life, but, you know, going forward, I was faced with this decision. Like do I follow the

recommendations? Which were basically take a bunch of medications and and eat a eat a processed food. It. Or do I? Take? What all I've learned and Implement those strategies and, and try and heal my heart in the way that I've learned is best. And, and I'm happy to say that, you know, that my, because this, I would have been able to talk about this at the last time. I we had an interview because it hadn't happened yet. But but yeah, I'm happy to say that, my three-month echocardiogram.

My heart function is normal. It's back to normal. Within like the ejection fraction of my heart is normal and the Damage that was incurred into The middle of the septum of my heart with a heart attack was was barely even present anymore. Like it, it completely recovered and I did that only taking one medication which was the blood thinner. Now that I had a stint in an artery.

It was a good idea to be taking a blood thinner for a while because we don't know how my body was going to react to that stent. But other than that, I didn't take any of the other medications and I implemented my own health or my heart healing strategies.

And and the doctor was pretty amazed that in three months, my heart was was he And functioning normally do you I mean was it like a specific Catalyst that you could kind of pinpoint and say this was likely the main contributing factor to causing the heart attack or was there just a summation of multiple different things? Yeah. It was it was a summation of things. I mean, type 1 diabetes heavily predisposes me and you know, I can't say that I took care of myself in high school when I was

younger very well. I mean, I had very high when sees, I didn't really understand the long-term repercussions in that at that time, so that probably In help, but also, I was I was under a lot of stress at the time and I got like, you know, it was it was the year of covid and elections and all that stuff. So all that stuff was getting at me which are things. I usually wouldn't pay attention to but since covid had this health related aspect to it.

I was I was kind of into it because health is kind of my thing, but also I got some very stressful personal news. The very beginning like two and a half days or one and a half days before the heart attack happened. Probably the most stressful days in my life. And and so I think that there's no coincidence there. But also I've been reading a little bit about a little bit about exercise induced heart attacks. And I and people who are already in an inflamed state.

So potentially I was with type 1 diabetes and all the stress. I was undergoing at that time. I also you know the morning I had the heart attack. I did a very intense workout, which was my usual routine, like my kind of, like, hit training,

you know. Just going to complete exhaustion in the exercises I was doing and I had a pretty intense workout took a shower after that and then had a heart attack and there are there is some evidence out there that in an inflamed State. Like if someone's in an unhealthy inflamed State and then they do that type of work out that it can induce a clock because, you know, my CAC score six months prior, which is the kind of amount of calcified plaque in the arteries of the heart was Zero.

There was no calcified plaque in the artery of the heart, and And the Interventional cardiologist that did my did my NGO and place. The stent said that there was pretty much no athletes grossest, in my coronary arteries. It was just this giant cloth that formed in the left anterior descending artery. So if I was to suspect things, it was all that that stress and build up the type 1 diabetes, that put me in this inflamed State and then I did that work out.

And I induce this this clot and, which was super unfortunate, but that's, that's the best that I can. I can think of in the best that I could find looking into that. I mean, you know, in the hospital they were all just like, oh, your your cholesterol cause this or whatever and but I, when I kept asking questions, they would just kind of dismiss it and and wouldn't really open up the conversation to it. But that's what I think. My best guess is what is what

happened? Were you just like was your skin just boiling blood boiling skin crawling when you're in the hospital and you're seeing what their standard of care is, and what, you know, I mean knowing what, you know about the heart and Your personal situation like were you on board with some of the things they were doing? Are you totally opposed to most

of it? You know, I wouldn't say I was totally opposed to, I mean, it's totally opposed to a lot of it but not all of it. Like, I wanted their opinion, you know, because I was just, I was just in this acute situation and and I realized that one of the strengths of Western medicine, is it an acute situation that can keep you alive? And so I wanted to know what they wanted to do after, you know, after they saved my Life.

And and but every time I would try and open up a discussion with them about, well, what about the side effects of that? Or is that the best thing for me? They would just shut down the conversation and it's say, either say I don't want to talk about it. They'd say this is our recommendation and we're sticking with it or they say, I don't know anything about that.

So I'm not going to talk about it because which is understandable, because if they give me advice on something, they don't know anything about it, you know, they're they're in a hospital setting, they're all worried about lawsuits, you

know, right. And so I kind of respected some of their answers, but it was still Discouraging to feel like nobody wanted to listen, whether it was their own personal reasoning, whether they want to listen, they just didn't care or it was like other things preventing them from listening because they were scared of lawsuits and that's how they've been trained. But it was just a shutdown of

conversation. That was the most worrisome thing to me wasn't necessary, cuz II know, Western medicine, strengths and weaknesses, you know, I knew what to kind of expect, you know, being in there, but it was just frustrating that there was nobody really interested in what

I had to say. And and it was it was Mike are you know, it wasn't like I was just some cookie cutter, heart attack patient, you know, like they prescribed me a medication that long term has been shown to cause insulin resistance of square of type. 1 diabetics has diabetic is probably worse than for a type to, you know, and so I was just like, why was that prescribed rather than this other medication that did the exact same thing that hasn't been shown to do that.

You know, like it was very clear. They were treating my disease and not me as a whole person, you know, and That was that was Troublesome. But you know, that's where that's where I had to, you know, rely on my knowledge to kind of say, okay. I'm not going to do this. I am going to do this, but then I realized that, you know, I'm a bit unique and then I've done all this research for the heart disease and everything.

And so I know this stuff but there's people out there who are going to be in the same situation. I was in and they don't know this. So I realized that, you know, yeah, I guess I really needed to put the information out there because it may, it may be life-saving for some other. People put them on a path that will make it so that they don't end up back in that situation.

Because if I had followed some of the recommendations, I feel like I'd be right back in and they were telling me to eat completely processed food diet. And that was the best thing to prevent heart. Failure, another heart attack and I just knew that was absolutely, not true. I mean, there was things like soy and fig newtons and and Sherbert. And just all these processed foods that I was allowed to eat on this diet and it was just it was pretty disappointing. Yeah, I know.

Understand that, I mean, my wife Crystal and she was at the hospital, you know, before, like when she drank it, all of her GI stuff, figured out. They were recommending for

whatever reason. They just, like default to really easily absorbed and digested foods, which always are heavily processed high in carbohydrate sugary foods, but it's like, why, why in the world of all the things out there is that your primary recommendation is kind of what they tend to default to. It seems Yeah, it's pretty crazy. Like I was you know, they're bringing me food while I was in the hospital.

I was there for three days but usually after a heart attack like that, they keep people for a week, but I was I was doing a lot better and I just asked to leave because I couldn't wait to start eating real food again and start my own heart healing routine. But yeah, they were bringing me food and I'm trying to, like, avoid the most processed food.

So I'm just picking it things, but then it wasn't really enough, you know, the things that were on my plate that I did want to eat it. I would just end up being really hungry. Sometimes I was just like, oh, I'm just going to fast because it's probably better for my healing anyways, and so so yeah, it was, it was a bit of a struggle and it was during covid. So no one was allowed to visit me.

I was alone the entire time as far as, you know, family or friends being able to come in. So, one of my, one of my friends did bring some stuff to the hospital and they brought it up to me, but I wasn't allowed to see anybody. So nobody could bring me food or or anything that I wanted. And that was the other thing too is that I'm Type 1 diabetic and and I have a very regimented way of controlling my blood sugar's at my age. One. Caesar, very good for a type 1 diabetic.

And despite, you know, seeing them, my agency was very good. They insisted that they would that they should treat my type 1 diabetes, the way they wanted it to be done rather than what I do. And they wouldn't let me control demonstration of my insulin and things like that. And so, my blood sugar's were very high. And I kept saying, look, this is not working.

Let me do it. I know what to do and they kept saying no, so when my friend brought me that that stuff and they brought it up to me, there was insulin and then I started managing myself and just said, no. I'm not going to take yours. It's maddening. It's crazy. And some of the nurses were even like they were even like, I don't know why, they don't let you type ones, just control things because it was under the control of like the pharmacist in the end.

The doctor the attending, there is the, but some of the nurses was just Like yeah, this happens all the time. I don't know why, they don't let type ones, just control it themselves, but it's because the hospital is liable. Like, if I start administering my own drugs like that, they're giving me then and if something goes wrong, then they're liable. And that's just how it is. When in reality.

I was feeling terrible because my blood sugar was over 300 and then at times because I just couldn't control it in the way that they were trying to and so, and they wouldn't let me do it. So but eventually like on the second day, I got that stuff and I was able to get it down, control the way I wanted to, but it Is maddening to me, you know, yeah, that's got to be

frustrating for sure. I mean, I totally get the liability standpoint, you know, I mean, it makes sense what they do and why they do it, but I wish there was an easy way to bypass that like have you just simply sign a disclaimer or waiver and then, you know let you do your own thing and if they just Monitor and if it's obviously working then what could they possibly have against it? Yeah. Well, it's just crazy that that in a place like a hospital where you think.

The number one goal is to get the patient's better bear. Whatever means necessary. Terry. There's all these there's all these things in place that are protecting hospital and the doctors rather than what's best for the patient. And that's just, I guess the legal world we live in, but it's just interesting to see that that that play out, you know. Yeah, it is crazy, man. So ideally people will not have the perfect storm of cascading negative events that lead to a heart attack like yours.

I've had several people on the podcast talk of ways, like people can proactively monitor the health of their hearts and their Arteries to hedge against that and some people are huge advocates for the CAC scan. Some people are huge advocates for look at the lipid markers and some people had dr. Gundry on not too long ago and he was talking about a specific lipid profile that measures inflammation. I'm not sure what it was called again.

But is there anything that you personally recommend is like just the best and most ensuring way to kind of get ahead of it and proactively monitor things to make sure that you're not likely going to have a heart attack. Yeah, and so there's an interesting studies that I talk about in my book that show. Well, they looked at, you know, the traditional risk factors that people measure for heart

disease. So this is like an LDL or a cholesterol panel or lipid panel or inflammatory markers, or high blood pressure or various things like that, you know, and they looked at those and they assess all the research that, that looked at their those markers ability to predict whether Going to have heart disease or a heart event or something like that. And they found that they were very inadequate at predicting.

Those things in this paper. So that was pretty interesting because I have my own set of things that I would want to look at and it does include some of those, some of those risk factors that are the more traditional type ones, you know, but I look at them in the context of other things. So if I take a lipid panel and look at that, I'd rather rather

than looking at levels. As of LDL, which I don't think are very relevant when it comes to whether or not someone's going to have heart disease or not. Because I just I just posted a study into the other day on my Instagram that showed that LDL level has no indication, whether whether someone's going to develop atherosclerosis in the arteries because they looked at a group of people and regardless of LDL levels. Some of them had like it had low

LDL levels. Some of them still had atherosclerosis and other ones, didn't, you know, so it didn't really matter. So there's that. But so when I Get a lipid panel. I look more like markers that are going to tell me more about the person's metabolic health because the person's metabolic health is going to indicate whether or not their insulin resistant or not. And so that's a big deal because there's always going to be

damaged a lot even artery. It's just normal wear and tear of the body, you know, but whether or not someone gets atherosclerosis when the the land the lining of the artery gets damaged depends on that lining ability to repair itself, and they Build it to repair itself, depends on insulin signaling. And so if someone becomes insulin-resistant, their metabolically unhealthy than they, the the lining of the artery can't repair itself as

well. And we end up with the body of doing something else which is depositing, cholesterol and minerals that are trying to patch up this damage. So it doesn't result in like a rupture of an artery or something like that and that's atherosclerosis. That's what that's the hardening of the arteries that we're getting. And so looking for someone's metabolic health and whether or not their insulin sensitive or insulin, Sent it when more important.

So for that on a lipid panel, it's the trigged HDL ratio. That's way more important and there's plenty of research suggests that that's way more indicative of whether or not someone's going to develop atherosclerosis or have a heart event or something like that. But and then people talk about all the different, all the different tests.

You can do, like looking at the particle size of the LDL and looking at the small dense LDL or the lipoprotein, a or oxidized LDL all these different Subfractions, I guess of these lipids and those are all interesting but all those things like it's not like the small dense LDL or causing after this grossest. It's the things that cause small dense LDL that also causes atherosclerosis and those are things that damage tissues in the body.

So it's damaging LDL and creating small dense ldls are oxidized ldls, but it's also damaging the lining of the artery. So it's not that we want to like the treatment should not be focused on reducing cholesterol. Because they're small dense LDL, should be focused on reducing the things that damage the cholesterol, which to me is things that cause oxidative stress.

So oxidative stress is just when there's lots of free radicals in the body that are that are causing damage to different tissues in the body, including the lining of the artery. And so we want to reduce those things.

And so for me, the first numbers you want to look at our metabolic Health, but if you're if you have poor metabolic Health that can lead oxidative stress, but then there's also different things you can look at at when it comes to blood work and things that are will measure your amount of oxidative stress in your body, but they, you can get kind of in the weeds with

the easy. Another kind of obscure little things that aren't normally taken, but you could take a GT, which is a liver enzyme that kind of measures oxidative stress on the liver. You can measure different things like damage to different fatty acids or damage to DNA or levels of glutathione or things like that. That all can give you an idea if there's oxidative stress, but Regardless of, if there's oxidative stress or not.

I'd still want to do the best, I can to reduce it and do things to reduce it. Even if it's, I'm not testing high for it, you know, because it's the oxidative stress and inflammation that you could do. You could look at to to assess whether or not there's going to be damage, the lining of arteries, that may overwhelm the ability of the body to heal that

damaged. So you could also look at markers of inflammation, like, you can look at various cytokines, but, you know, the best General marker of inflammation is like a heist. Sensitivity C-reactive protein which is which is just definitely something that you know, cardiologist do. Look at when you look at like a cardiac pain all day. They measure that for sure because that's definitely associated with with higher risk

of heart disease and things. But then, I think the thing that is most underutilized as far as risk for heart disease is heart rate, variability. That's just something that you really don't find measure too much within Western medicine. Is there a heart rate of It heart rate, variability that you like to see kind of as a where's the line drawn between this is good or this is bad in your mind. Well, so there's really so anywhere from like 20 to 100,

is, is normal. So there's really no, like, like, with heart rate variability. There's really no, like, you don't want to sit there and compare like someone yours or someone else's, you know, like if yours is 80 in someone else's is 30, you know, those both are normal. You really have to kind of track it over time and find out what's normal. For you. And what's what's your kind of average is so that you can see when it changes, right?

Because when it's all kind of relative, I guess, because if I if I sit at say 40 normally and then I'm tracking it and it drops down to 20. It's like, okay, what happened? That's not normal for me. You know, that's kind of how I tell people to look at heart rate variability, because you can't really just say, oh, I want to be in this range and In the last chapter of my book. I kind of talked about how all these different ranges, for different things.

On labs, are different biomarkers that we have may not actually be as accurate as we think. I mean, the ability of I Define health is the ability of the body to adapt to certain situations. And so trying to trying to quantify Health in a way that puts everything in little boxes and it says, okay, it has to be between these numbers is kind of kind of backwards when we look at what what Fizz because health is like the ability of your body to go out of those ranges and come back, right?

You know, that's that's what health is. And so just like, you know, if you're if you like health metabolically is if I eat a ton of carbohydrates and I take my blood sugar out of you know, what could be considered healthy, it does it come back easily and get back into ketosis? After I do that. That's metabolic Health, you know, and so just so if I ate a bunch of carbohydrates and I My blood sugar and it was too high. So she's like, oh, that's unhealthy.

Like well, no, that's what the body supposed to do move. It comes back. That's how you know, or or adaptation in the in autonomic nervous system. Like, can I go through a stressful thing, a stressful response and come back to Baseline, come back to balance and the autonomic nervous system that's held, and that's what heart rate variability is kind of measuring.

And so, if your if your heart rate variability is kind of in this narrow window and it's lower than you don't have the best ability to adapt to a stressful situation, which can Too bad things happening in your body, you know, so so yeah, it's interesting, you know to look at things like that because you know blood Works never going to tell you if you're healthy. I'm not saying that we should never take it or anything.

I'm just saying that you know, there's I know I've had plenty of people who you know, come to me with blood work that is completely off. So to speak. Like things are way out of whack everything, but they feel amazing. They have no symptoms whatsoever. And then there's people who have all kinds of symptoms and their blood looks Well, work looks perfect, and they're just looking for explanations and nobody can give it to him. So it's just this, it's this

tool. We use what we shouldn't, really use it, or should we allow it to tell us a healthy? Not we just use it as a part of an overall assessment on of our health. But yeah, that's those are my kind of things that look at someone's metabolically, healthy. Look at the amount of oxidative stress and inflammation and then definitely balancing the autonomic nervous system by using heart rate, variability, and make things better. You just using like a or ring or

something for HIV. Yeah. I that's what I use to. Acts that, but there are lots of different things. I mean, I don't know how effective, a reliable, like the Apple watches are, but they'll do it. But also, there's like a lead HRV, there's different devices. People use like the Bayou straps and just like something you can put on your wrist. So there's lots of different devices that will help you track

that. Yeah. I've got the word ring and I've been kind of keeping down HRV, my wife Crystal. She's pregnant right now. So her HRV is totally plummeted to like the teens, which is much lower than normal, but I'm assuming that's probably normal in the context of being. Pregnant, but mine's consistently right around 50 or 60, which is pretty normal for me. I'm probably sleep as much as I should. But when it comes like that the lipid profile, ma'am. I don't have any markers or

anything. That would be concerning. Like, I look at mine. I'm consistently seeing Triggs below, 50, H or HDL, you know, normally north of 100 and my LDL, this is something that I'm curious about LDL seems to fluctuate tremendously based. Of what people consume the night before they get tested. So if they go in have like a, you know, particularly fatty meal saturated fats, barbecue for instance, and then do a 12-hour fast and go get their

Labs drawn the next day. Like that's typically going to result in a significantly higher LDL count, which alarms their their doctor, their General doc. So it just, it becomes a very skewed glances to their health because it fluctuates tremendously based off of something that, you know, they had the night before. Yeah, definitely.

And and yeah, and you know, I'm sure you've heard of like day Feldman and he's shown that, you know, if I if you if you don't fast long enough then some of these numbers can be off. So even a 12-hour fast may not be good enough. If you're having, you know, High animal protein, high fat type of meals, especially for triglycerides, but also for LDL, so it's important not to say, you know, at least 14 hours for

some people in that situation. If you if you want to, you know, get Truly accurate numbers because lots of times, you know, those these more like keto meals and heavily more heavy animals. They take a little longer to digest, which is not a problem. But you have to account for that when you doing blood work, you know, right. Right. I think. I'm not sure what podcasts, I listen to, I think it was saladino on Joe Rogan or

something. He was saying that, he's got LDL north of 500. I want to say, is there a point at which, like LDL exclusively being incredibly high as alarming? Or is it all just It's part of the bigger picture. I mean, I think the jury is kind of out on that. I haven't seen anything to suggest that having higher LDL like that high would be problematic. But that doesn't mean that it may be more of a result of nobody's really looked at it. And I think that there are people.

Alright, right now, out there trying to design studies to look

at specifically that. But in my opinion, I don't see how it would be, you know, if there was if there, if someone was in a very inflamed Aid and had lots of oxidative stress and having more LDL around, could be problematic, maybe it would, maybe it would lead to, you know, less or higher viscosity of the blood, which is more likely to induce clotting when the Bloods just not moving as well as it should be or or maybe maybe the things that damage LDL, there's more LDL to be

damaged, you know, and then that damaged lbl can contribute to damage as well. It's just like this Cascade of events in is So like maybe in that state, but if someone's, you know, living a pretty healthy life that got low levels of information low levels of oxidative stress and I don't see why that would be an issue. And, you know, in my book, I talk about how there's this layer of fourth phase water that surrounds, the inner layer of the artery.

As long as we're putting ourselves in the right environment, which is contact with the earth and sun light and infrared, light and things like that. And so if that layer is intact, it excludes everything, that's not it. So it really doesn't matter. How much LDL is around. It's not getting to the lining of the artery if that layers attacks. So it's all about putting yourself in the right

environment. So yeah, so, you know, we'll see in the future, when some of this, these studies come out, but I based on anything I look at I don't see it being an issue. Now. There is some studies and people with familial hypercholesterolemia, which is where they haven't genetically very high LDL levels. And it doesn't seem that they live any longer or shorter than anybody else.

And one of the studies said that, The people with that condition who did live shorter, it was likely due to other lifestyle factors, you know, they were smoking or drinking and not exercising, or eating processed food diets. Like it was those things that cause them to live a shorter life, not their higher LDL. So so, yeah, I don't think that

it would matter, but we'll see. And when other research comes out, you know, I think so much of it has to be kind of viewed through the lens of, you know, if someone's following a ketogenic or lower carb, higher fat, based diet versus more of a traditional standard. In your mix Diet and then I would feel like the markers that you're getting are, you know, by definition going to be highly skewed.

So you can't really, you know, approach the treatment necessary for the individual through the same lens. I mean, I'm boggled by people that go and get their their lipids done and in their their doctor automatically prescribes them, a Statin without any question whatsoever as to what their nutrition is, like. My dad just got put on a low dose that in his, his ldls, not even 200, like it's not astronomically. Any means, but like, that's not even part of the general conversation.

When you go to the doctor and they automatically prescribe, you stands. It seems. Yeah, and so, it's interesting because if you trace like kind of the history of what the recommendations are, what LDL should be or what cholesterol should be.

There was a lot of meetings in the past that were specifically set up and funded by pharmaceutical companies to lower the LDL recommendation, you know, to lower what it should be. And you know, that's more or less because the Pharmaceutical companies know that if the recommendations is it for LDL to be under 100, in total question will be under 200. They can prescribe more statins. And so you have to take into account but there's that

Financial backing there. But it also tells me that we really have no idea what LDL is supposed to be what is ideal. And maybe there is no ideal for any blood marker and that's kind of the philosophical question. I try and discuss in the last chapter of my book is that we really don't know because, you know, if we take, you know, where Menswear for millions of years which is out of out in nature, eating a natural food diet, whether it be, you know, more plants or more animals or

whatever. It was, whatever was available to them. You know, which to me was probably more animals. What are their blood work look? Like, you know, like the normal is that we have for our blood work are based on, you know, healthy populations within the context of a modern-day society, which is nothing like what for the majority of time humans. Modern humans have been around. We're living a Z, right? So so how do we know that?

You know, that those ranges are what are actually normal for humans or if there even is a normal. I mean, there was a study that just came out recently maybe maybe six months ago or so. Then I talk about my book that that that showed that like it's just an association of study. So you have to take it with a grain of salt, right? But it's it showed that the levels of LDL and levels of total cholesterol that were most. Released associated with any type of heart disease or

all-cause mortality. In general were a total cholesterol between 200 and 250, which is above what they say. It should be in an LDL between 100 and 150, those people who had those levels. There had the least all-cause mortality, at least heart disease, cancer least infection all that kind of stuff. So, so, and that's just an association so we can't really, you know, say anything from it, but like it's just interesting because if LDL and total cholesterol was so causative in heart disease.

You'd think there would at least be an association, you know, between having higher amounts and and and more disease, right? But when we don't see that, it's almost like, if these Association studies not seeing an association is, is way more useful than seeing one and saying, oh, that's the cosmic because you can't really prove that, you know, so it's just interesting to think about that. You know, we humans are trying to quantify things. That that create disease and

measure those things. So we can assess our risk, but even like some of the major editors of the major journals, like Marcia angle, who is the editor of the New England Journal of Medicine has come out. And said things like, you know, these, these medical journals are being used heavily by pharmaceutical companies as advertising, you know, so it's basically, yeah, it's basically what can we, what kind of study can we design to?

Show that our product is useful or that, you know, higher levels of this are problematic so that we can make a drug to bring it

down. You know, like it's they're being used in that way rather than just purely asking questions about science and finding the answer, you know, like it's pretty scary but it just means we have to use that stuff as part of our, our toolkit, you know, to think how we should live and how we should be healthy and everything, but always keep that kind of stuff in mind. Yeah, would it be? My mind how like you talk to anybody in the general public and you mentioned the word

cholesterol. They automatically associated with a negative thing that is is supposed to be lowered, you know, as much as possible. I think of like cholesterol being the precursor to testosterone and every cell in our body having, you know, a layer of fat surrounding like you don't want to reduce cholesterol to the point where it's insignificant and I feel like a lot of people are doing so and their health is not improving as a result. I mean there, I don't really know.

So to the extent of which, this is true, if it's true at all. But there has been some studies that show, you know, potentially adverse effects from too much Statin. You try it from like a neurological standpoint. Is there any, is there any weight to that? Or is that not so much the case? So, definitely. Yeah, so I go through that and chapter 7, my book, like, you know, not only all the benefits of cholesterol and what the body

uses it for. But also all the side effects of statins, you know, there's I just saw a study the other day. I'm with someone I follow on Instagram.

Newer study. That was showing that, you know, keeping your LDL too low like, you know, some cardiologists, like we want it below 70, you know, they aggressively use statins and then it, basically, the study showed that people who did that had had cognitive issues because the brain is largely made up of cholesterol, you know, and so but there's also, you know, like making making different hormones, making vitamin D. It's different precursors in the

in the process of making cholesterol are used for different things like making An internal antioxidants, which are really important for reducing oxidative stress and inflammation or, or insulin signal. Or it's so much chapter 16, you know, one of the precursors is used for that which, which we want to prevent insulin resistance, which we talked about already. So, like, cholesterol is useful for all these different things.

It's necessary for all these different things and if you lower it to aggressively, you know, you're going to have issues. And that's why that one Association of studies. So that that, you know, LDL between 100 Was associated with the least all-cause, mortality and disease. No, because your body is using it for these things. And so, you know, there's plenty, plenty of evidence that I go through, in my book about how a statins are not preventing heart to heart attacks or heart

disease. And in some cases, they're even creating more like atherosclerosis and things. But also that they're also causing another set of symptoms. I mean, I'm a I'm a chiropractor. I do miss them only mainly like neuromusculoskeletal in my clinic, you know. And I can't tell you how many people have come in and said, you know, I'm at started having this pain and I asked him. Okay.

Well what what changed? You know and and lots of them, not all of them obviously, but lots of them say, well, I started taking a Statin, you know, and it's causing muscle pain because cholesterol is essential for for muscle function. And so if you'll owe her too much, you're not going to get proper muscle function.

It's going to be painful. So yeah, so all kinds of things, all kinds of issues and that's just the I got to just highlight the The I guess the shortsightedness of of the way pharmaceutical companies and Western medicine, kind of thinking about things. It's like rather than make the body work better or figure out. What's why it's not working. Well, let's try and change one biochemical Pathway to get a result on a blood work, which is you treating the blood work and

not treating the person. And so, and then yeah, then you're then you're creating all these other issues because you're changing one biochemical pathway in a very complex biological. Logical system that is not that's not just going to affect one thing. It's going to affect all kinds of different things in that complex system. Do you, do you check your blood pressure regularly? Like do you take that into consideration through any of this?

No, not really. I mean, I I mean, I've always had relatively normal or low blood pressure not like pathologically low. Gadget or anything like that, but so at that's always been fine, but even, but even then, like blood pressure is like, we're again, we're very obsessed with keeping it below. 120 over 80, you know, but, you know, your blood pressure going up, is it is an indication that your body's trying to adapt to something to some situation.

You know, that's why there's white coat syndrome when you have high blood pressure. When you go to the doctor because you're nervous, right? Like if your blood pressure didn't go up. When you were nervous, I'd be concerned, you know, and so we're getting rid of obsessing over keeping these things where they need to be. Rather than recognizing that the body needs to be able to attack the different situations. Now if it tries to adapt to it

and it doesn't come back down. Okay, that's an issue. You know, like if your blood pressure stays up when you're when you're just relaxed at home or whatever, under no stress or whatever. Then okay, maybe that's an issue or if your blood sugar, you know goes up and never comes back down or comes back down to slowly. You know that okay, that's insulin resistance.

That's that's a problem. But people get really obsessed with these these numbers and what they're supposed to be and and Instead of measuring the body's ability to adapt which is taking these static numbers on blood work or Biometrics that make sense. Yeah total makes total sense when it comes to you you're going to have probably a little bit of a different situation because you are type 1 diabetic.

But when it comes to what, you know about the heart, how it functions always interlinking, you know, equations when it comes to your nutrition, are you you're pretty much following a ketogenic low carb approach, right? Yeah, it's pretty low carb. I so I like, I like to say that I follow metabolically flexible die, which could be a lot of different diets, you know, because you could be, you could eat a vegan, metabolic inflexible diet.

That's possible. You could eat a truly ketogenic diet were seventy percent of your calories, come from fat and that could be metabolically flexible. But the idea is you want to keep your body able to burn fats and

carbohydrates readily. At any time, you know, so if you and generally that means lower carbohydrate, you know, and not having tons of carbohydrates all the time, doesn't mean you can never have them, but you want to keep them to a minimum and you want to make sure their whole food carbohydrates, but the majority of your energy comes from fatty acids, and I think animal fats are way better than plan fast.

And I talked about that in the book, you know, having cholesterol versus 50 straw, which final straw was like, basically the plant version of cholesterol, and why Having cholesterol is way better for you than Phi toaster all. But, but yeah, so I think that, you know, just eating in a way that creates metabolic flexibility. And so, for me, that's, you know, pretty low carbohydrate, especially trying to control type 1, diabetes.

It's that's really useful for me, just to, to lower the amount of food that I eat that do raise blood sugar a lot, which are carbohydrate-rich foods, but I do eat, you know, some berries and and maybe a sweet potato every now and then think things like that, but generally it's Pretty low carb, very centered around animal foods and then, you know, lower carb vegetables and things like that. That's that's pretty much what I

eat. Yeah. Yeah. I mean most people following a standard American diet or just any the outside of a local cover approach are never really given their body opportunity to become fat adapted. So, you know, once you go that route once you go, you know ketogenic even if only temporarily your body builds up, those metabolic pathways to A function and fuel itself with ketones and fatty acids, which is advantageous. Do you think there's any like

for me? I don't introduce a bunch of carbs in. I mean, I don't feel better when I do. I don't perform better when I do but I don't also think that my body forgets how to metabolize glucose. So if I was to eat a sweet potato right now, my body wouldn't know what to do with it. Like it's not like it forgets how to do that. But most people never become fat adapted to begin with apart from when they're born.

So they would, they would benefit from from having these periods of switching back and forth. So to speak like you're saying with regard to the metabolic flexibility. Yeah, it's really interesting because you can like metabolic metabolic flexible is way worse. When you become to align on carbohydrates. It's and if you're eating the standard American diet and having tons of carbs process carbs, especially all day long, your body forgets, how to, you know, metabolize fats.

It doesn't forget, but it down regulates those Pathways and it, it almost forgets how to make ketones, you know, and so when you start going lower carb, that's why there's like this to 26 weeks, period, where you have this, you know, keto flu kind of thing, because your body is really having to work hard to update. Those Pathways again and teach itself to to, to burn fatty acids, and make ketones for fuel and stop using carbohydrate now, but you can become metabolic

inflexible. The other way, if you stay in ketosis for a very long time, but typically, it's like, you start E2 carbohydrate and, and they've shown this. Other people are in ketosis for a long time and then they eat a carbohydrate, their blood sugar does go higher and it looks like they're in so a distant. But if you did that for like a day or two, then your body starts burning this carbs again here and you're just fine. So it's just like the time period. It takes two to get metabolic

effects. Will from a high carb diet is way longer and way more damaging, I think because burning glucose primarily from fuel creates more oxidative stress and less ATP and things but also it just takes longer to get there. Whereas, you know, if You were in ketosis for a long time like your body goes back to burning carbs pretty quickly. That it's not going to cause any issues. You know, 100%, 100%.

Yeah, I've had the you know, some people have raised the question as to if it's damaging to be low carb ketogenic, you know, except for extended periods of time. I just I don't see any any indication that that would be negative. Like there may be that, you know, 24 or 48-hour period in which you have, you know, hired blood blood glucose after a postprandial meal with a hiring carbohydrate, but Like you said that that would typically level

out after just a few days. Yeah, your body would would readily go to burning those carbohydrates. Again. Pretty pretty easily. Yeah, when it comes to, you know, with regard to nutrition, you were talking about the plant sterols versus, you know, animal fats. I'd love to kind of dive deeper net. So, you're more in favor of saturated animal fats than just loading up on a bunch of monounsaturated fats or certainly, highly inflammatory polyunsaturated fats, correct? Definitely.

Yeah, so and it's and it's not necessarily just polyunsaturated fats, it specifically plant versions of them, you know, because like when I think about but you know perfect fatty acid ratio makes a mistake, you know it. Yeah, it's a just like, you know, grass-fed meat especially, you know, like that's what we want.

And the problem is that we don't it's not that we don't have enough Mega threes and we should pop up like Jamaica three pills, you know, it's it's that we have way too many Omega sixes because people are eating vegetable oils and things like that. So that's the problem. So rather than try and even have to ratio by taking a bunch of Omega-3s or eating a ton of Mega three, which reads we should really just lowered the omega-6

foods that we're eating. But even even beside besides the omega-3 versus Omega 6 discussion. There's cholesterol versus Phi toaster, all which there's, you know, cholesterol is made for Animals. It's the fat made for animals and phytosterol is made for plants as well. Plants use. It's like the cholesterol version of for plants, right? And so, We're animals, right? Yeah, so we use cholesterol.

That doesn't mean that our bodies can't use my toaster all in place of cholesterol that has to, but it would much rather use cholesterol. And there's actually some studies that show some evidence that, uh, you know, when we eat lots of phytosterol, which is like vegetable oils and plant fats, and things like that, that they compete for absorption in

the gut. And we, absorb less cholesterol, which is why the, which is why the Like things like margarine and vegetable oils are touted as Hearts help our heart healthy because it had it prevents absorption of cholesterol. Therefore lowering cholesterol in the blood and so they said oh that's heart healthy, but you know, the fact that lowering cholesterol is not heart, healthy was never taken into account. But so it's interesting.

But when your, when your body like, when we're eating tons of these phytosterols and your body is forced to use, use that in place of cholesterol. I talk about some studies in my book that That are that are the looks like it's pretty problematic. Right? So one of the one of the issues is that you know, the red blood cells look like kind of like a donut in their shape.

They they use cholesterol to build up the cell membranes and that makes them very flexible makes them able to bend around. So they get into those very small capillaries. They can kind of squeeze through, right? And sometimes, they even been themselves a little bit to get through. However, if our body is forced to use Phi toaster all to make those Get very rigid. And so then they try and squeeze through and they end up damaging

things. And so they've done these studies in rats, not in humans, but they've done these studies where they feed them more phytosterol and there they definitely have more Strokes that caused more damage to these these in arteries, the capillaries because they can't squeeze through as well. And so I want to build my red blood cells with cholesterol, not with Fido straw.

And then, there was also this other study where or it was actually part of the same study, where Are they they did surveys on people who were going to get a heart valve transplant.

And they asked them how many or how much they basically asked them like, food surveys or whatever to give an idea of how much phytosterol, how much plant fat, they were eating versus animal fat and it showed that the people who ate the most plant fat when they went in there and they replace the valve, that the valve, they were going to replace had way higher amounts of Deposited fight. Oster all around the valve.

And so, and the one who reported eating lab s and was a direct correlation eating less had much less deposition of phytosterol around those valves, which is interesting. Again. It's kind of Association, but it's a pretty interesting Association. So it to me, I just think that, you know, where designed to to eat cholesterol and when it comes to unsaturated fatty acids, we're supposed to get those from animals as well.

You know, it's not These plant versions aren't serving us as far as their hiring a mega sixes, which we want to keep that down a little bit. We still need to make a success but keep that down to a healthy level and also their phytosterol, not cholesterol. So basically avoiding all the soybean oil grape seed, oil grape seed, oil flaxseed oil, all of these plant-derived fats and oils that are are not really

going to be boding. Well for our the red blood cell, basically the formation of red blood cells. Yeah. Yeah. That's those. That's the big issue because Like I'm not going to sit here and say, oh don't eat coconut oil, don't eat olive oil, like pure olive oil. You gotta make sure it's pure, although not contaminate with coconut oil, or, with canola oil, or things like that, or like avocado. I'm not going to say, Don't eat. Those are definitely healthier things than the standard

American diet, right? The real issue is to the widespread consumption of the vegetables. That's what's driving up. The amount of phytosterol were eating. Its what's driving up, you know, oxidative damage to those things. It's what's its that's the real problem. And so we need to focus. Asan, animal fats. And if you want to include some plant fats, like coconut oil or olive oil, things like that. That's fine.

But your diet should have lots of cholesterol and animal fats in it to make sure that that's the primary thing. Your body is using to build, not just red blood cells, but also is really but especially the red blood cells. Totally. It's been interesting to see

minute. I've been in the space longing to see how these different types come and go and, you know, for a while it was like blaming sugar and sugar probably obviously not optimal but then Lately, it's been this this massive finger pointing towards highly inflammatory polyunsaturated, vegetable oils, you know, and they've, they've looked at

graphic graft that out. And so on that, you know, the Obesity epidemic is kind of more or less correlated to the same Trend and increased consumption of these oils. So it's like people are trying to say that's the primary driver and maybe it is. Maybe it isn't not sure but you get other people in the space not so much the keto space but other nutritional spheres that are saying. Look there is no issue whatsoever with these you know vegetables eat. Eat them at libitum.

Don't worry about him at all. And I'm just amazed at how much conflict and controversy there is out there. Like, how come there's not just some Standalone like we can agree on this not being optimal, but that doesn't seem to be the case. Yeah, and I really like, I read 10 Naamans book and I really like his approach because he breaks it down to, you know, we can sit here and argue about whether or not these these oils are good or bad and we can design studies that isolate different.

Balls and try and show that though, the unsaturated fats aren't bad or whatever. But I like his approach because it's simple. It's like, you know, when you eat a food, you know, you wanted to have like, you need, you need energy, and you need nutrients. And so the energy either food that has energy and nutrients and that's good. Right? So, like a steak, it has energy in the form of fat and, and it has nutrients that sounds of protein vitamins and minerals. And so that's a good food.

Right, but then you take something like a vegetable oil. That's just straight energy and that's not good. That's not, that's never going to satiate your body because it needs energy and vitamins and minerals. So I really like that because it breaks it down. Like that's just logical. It makes complete sense. Like, and you don't have to go into the Weeds about, you know, does it? Cause oxidative stress? Does it cause heart disease? Like what like does do polyunsaturated fats from plants?

Like do they cause insulin resistance? Like, which I think they do but, you know, like you don't have to do all that, you have to say like That's not a natural food. That's not what humans have been eating for millions of years. Is this this refined vegetable oil or refined sugar like anything? Those are just straight energy. There's no nutrients within which is a problem. Yeah. No, I like to name is approach

for sure. I've had him on the podcast several times and we have a lot more common ground that not people want to inconstancy. He's on one in the Spectrum on the other but there's a lot more common ground than not.

And I feel like people people in general, just like to operate in the extremes and people like to do this, incredibly high fat approach or this incredibly A high-protein approach and that oftentimes just simply not sustainable and obviously, you know, whether you're consuming in a surplus or in a significant deficit is going to have an impact.

I mean if you're consuming at a massive Surplus and that Surplus is comprised of a lot of Highly inflammatory foods, it's that's certainly not going to bode well to improving your overall composition when it's all said and done. I mean, you got to take all these factors into consideration. Definitely.

Yeah, and it's, yeah, the extremes kind of people stuck in it and I understand it too because you know, it's When you, when you file an extreme thing and you limit everything else, it it it kind of helps you stay on it, you know, like you can't say, oh, I'll have that every once in a while because you getting a end up eating a lot of it, you know, I can understand for some people that by doing an extreme thing. It helps them stay more adherent to whatever they're trying to do. Totally.

Yeah, I get that. But yeah, sometimes I think it's taken too far and we get dogmatic about it, which is a problem. And so, so it's better to be, you know, More open and in your thoughts and things like that. Yeah, I mean for when it comes to nutrition, I probably just I try to just make it super simple and tell people. Hey look, you're going to benefit from having periods of time in the caloric Surplus. You're going to benefit from having periods of time in a caloric deficit.

And if each of those times you make that food consists of the highest quality, you can, you know, from good singling reading, Wholesome foods with proteins and fats. You're probably going to be all right, and if I mean that that's the huge oversimplification, but if people simply just did that, they would certainly be doing You know, classic 80/20 analysis, and proven things for the general part. Definitely. Yeah, so what do you got coming, man? What's in the pipeline for you?

I am obviously speaking of Kita akan. Nice. And yeah. Yeah, I'll be there in July, the book comes out. So my book was originally self-published but I got picked up by a publisher and so it's being re-released with you know, I added some things. It's information that I wanted to add and end. It comes out April. 7th. It's available for pre-order now, so that's the big push right now as we're trying to let people know that that's coming

out. And you know the book it talks about a lot of things that about the from The evolutionary origins of heart disease and like the evolution of mitochondria, all the way up to like the diet heart hypothesis and how that developed with the ancel keys and everything. But it also talks about, you know, how the heart's, not the main mover of the blood. And that's not why it's there, and the mechanisms, by which the blood moves.

It heart talks about why heart cancer is rare and the specific characteristics of the heart that that make heart cancer, very rare, and you know what, I think of the true Has of atherosclerosis, you know, everything from the relationship of the health of our mouth to heart disease, to to our social aspect to the autonomic nervous system. I go into a lot of different aspects of Health, but definitely hard health. So, that's the big thing for my nose. At the books, coming out, April

7th. And you know, I'm always I'm always working on new things. I mean, I'm in the very beginnings of may be gathering information for the next book. But but yeah, that's what I got going on. Now, nice man. Well, I'll be, I'll be getting a copy. When that comes out for sure, what is, what is your website resource? Your health.com resource yelp.com. Awesome. One more thing. I meant to ask this earlier, but I totally spaced out the past two years. We've been hearing a lot about

myocarditis. I know she had a blog post about that on your site. What is that? Exactly? And what have you learned about it over the past two years? Yeah. So there's there's myocarditis and there's like pericarditis. So myocarditis is actually inflammation of the heart muscle itself. Cough.

And it's just like, when this inflammation happens in this pericarditis, which there's like this kind of like the sac that surrounds the heart that kind of isolates it, you know, within within your body and that can become inflamed as well. And you lot of people talked about that and how it develops. And you know, there's people have been saying that it's developed because of the vaccine and stuff, which I wouldn't be surprised if that was the case, but I don't know if that is the

case or not. But various things can can relate to this. And it's all we've talked about inflammation and oxidative stress and over this past hour and the things that cause that inflammation oxidative stress or going to cause inflammation anywhere in the body, if you happen to be susceptible in the area of your heart, like the heart tissue or or the tissue that surrounds the heart that the pericardium, then, yeah, you can get inflammation in that

area and the issue with that. Is that is that that like, your body is put together a very compactly. And there's no room for things to get inflamed and larger and things like that. It's Why its my carpal tunnel is an issue because that nerve gets inflamed and that carpal tunnel is put together very compactly. And so if something is inflamed, it starts to cause pain. There's no room for that to happen. So same kind of thing in the

heart. And so what ended up happening there is if we get inflammation like that then the heart can't

function as well. You know, it can't contract like it's supposed to because there's this inflammation pushing on the side and so that can lead to that can lead to fluid around the heart, which can cause you because that's that's pushing into the heart and and Preventing it from Contracting, which all this stuff will end up like the heart could end up like kind of reshaping itself a little bit.

So it's instead of the end of it being shaped more like a football, can be shaped more like a basketball. And that's what we call it. Like, you know, dilated cardiomyopathy, where we get a car failure, which basically means that, you know, the heart is not functioning as a supposed to, I have my opinions on what the hearts function actually is, which I outlined in the book and what Ash Drink its blood moving. But if we're not doing those things, then we get information

of the hearted. It changes the function, the heart can't Vortex. Like, it's supposed to it. Can't move blood through itself. Like especially we have to get pulling up of fluid, which is, you know, a demon like in the in the arms and legs and things like that. And and so the but the treatment, you know, is we need

to reduce inflammation. We need to get the blood moving in other ways, which I talked about like as far as like being in contact with the Earth and and infrared sauna and things like that infrared light in general. But also, we need to remove the things that are causing the inflammation, which could be environmental toxins could be stress. Could be poor diet, those types of things and environmental toxins.

I think are the big ones, which I go through, like, you know what, those could be. And where to look at in your life to reduce those exposures in the book. But yeah, that's that's the inflammation of the heart. Everybody's been talking about and that's been shown to occur in certain cases with both the virus and the vaccine.

And it's not like it's targeting the Art specifically it's causing inflammation everywhere, but it's more pronounced or more noticeable in the heart because of that compactness. Yeah. Well, yeah, I mean, and it really just depends on where someone susceptible to it. You know, like was there was their heart tissue already mildly inflamed because they were on a poor diet. And then that was just the straw that broke the camel's back.

Like I don't know. But you know, any any inflammatory response to something leaves us, prone to those types of things. You could have inflammation of any tissue in the body, really? And and it's just this and some people like it tends to be the heart and and the heart is a place where there's a lot of friction, right? Because the heart is Contracting. It's moving all the time. And so if you are more prone to it there and you inflame the body and then you have this

friction as well. That's just going to perpetuate the inflammation. So the best insurance for anything. Anytime everyone is to minimize information whenever and however, possible and improve metabolic Health, however, possible as well, basically, Yeah, and, and, and, you know, also keep your blood moving as well as possible, you know, and so I talk all about that in the book how how the heart is not necessary this pump.

We think it is. It does some pumping but it it, you know, it's not the main mover of the blood. And so there are things we need to be doing that help blood, keep moving because that's going to take a lot of pressure off the heart. Take a lot of friction off the heart. So if they do get into the inflamed State, the heart's not having to work so hard. And then therefore creating more pressure and friction.

And so that's things like, I mean, I just did the infrared sauna this morning because the clear benefits is so many different benefits for it are from it. But but that's one of them is that it keeps blood moving very effectively. So don't get stagnant. Blood pulling up in areas and more prone to, you know, clotting or inflammation of things like that. I know you. Look at HRV quite heavily deep. Do you put much emphasis on just simply heart rate in general?

Not necessarily. No, I mean again that is something that I think that, you know, like, we're designed to adapt if your heart rate doesn't go up in certain situations that's concerning, you know, in general like heart rate. If it's if it's high when you're resting that can indicate that your heart is working too hard for whatever reason and and for me, that's because it's being forced to act more like a pumpkin. It's supposed to be. So it's having to work harder and faster.

So we can do things to take that. Pressure off of it. But in general, like, you know, what happens, like measuring your ability to adapt heart rate to go up and then come back down is way more important than just what it's just static heartbeat, you know. Yeah, I think that's a way better measure. But yeah, heart rate, heart rates important and obviously, like, highly trained athletes have lower heart rates because they're the, they become very efficient.

But I also think about what do highly trained athletes. Do they usually outside more, you know, they're usually doing Things that that helped take pressure off the heart, they're not just using their heart more and training it. They're, they're also using doing things that keep the blood moving so that the heart doesn't have to work as hard, which is why it's probably lower. Yeah, so, you know, getting getting the blood flow and getting some activity and

working out. I mean, all these things like it's funny like we do all these deep Dives and in nutrition and you know, heart health and lipid panels and everything and then like the resounding consensus always seems this be just eat healthier. Move more and, you know, get out in the sunshine, get some sleep, like all these super basic principles that if people just prioritize them their their ailments would likely be significantly reduced.

Yeah, exactly. And unfortunately many places in our many people in modern day Society, just those things aren't prioritized because of the lifestyles that we've created for ourselves. So it takes some work to prioritize them for sure. Yeah, totally. Well, man. I am always learning something like to talk. When I talk with you. I'm super excited to be the Look, I will definitely link out to that your website, your social profiles.

You are a wealth of knowledge on so many different levels, man. That's always pushing each other podcast. If there's anything I can do for you and just let me know. Hey, man, thanks for having me. Take care, brother.